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Epigenome Modulation

Epigenome Modulation Process

The NG Epigenome Process may be used as a stand-alone therapeutic process or in conjunction with other complimentary or supplementary NG Processes. It is frequently combined with the NG Full Spectrum Brain Integration Processes and/or the NG Anti-Aging Process. Additional information on the NG Epigenome Process can also be found at

Epigenome Definition:

Cellular and physiological phenotypic (physical and biochemical characteristics) trait variations that are caused by external or environmental factors that switch genes on and off and affect how cells read genes instead of being caused by changes in the regular DNA sequence are the work of the Epigenome. The Epigenetic concept seeks to describe dynamic alterations in the transcriptional (building block) potential of the cells. Such alterations may or may not be heritable. Unlike genetics based on changes to the DNA sequence (the genotype), the changes in gene expression or cellular phenotype of epigenetics have other causes, thus use of the prefix epi- (Greek: επί- over, outside of, around). The term also refers to alterations or functionally relevant changes to the genome that do not involve a change in the nucleotide sequence. It is also important in an understanding of the epigenome that it is highly responsive to sensory data input and its subjective (e.g. personal or client/host/subject) perception and mental/emotional interpretation. Our chemistry and neurology not only supports our existence it is also highly susceptible to it. In other words our personal attitudes, perceptions and unconscious programs have, as a rule, a much greater effect upon the epigenetic expression of our genes than any other factor.

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Interestingly, our attitudes and their expression—which serve as key inputs to the personal epigenetic expression of our DNA—are also highly affected by the same or similar aspects of our lineage, some of which can even be traced back to Adam and Eve and the roles and propensities of the man and the woman as they work out their joint contracts.

Epigenome Modulation Process Steps

  1. Pre-Process Steps & Instructions

  2. Optimum Process Flow Application Intelligence Request

  3. Call to the Energetic Direct Organ/Circuit Activation/Modulation Utility (EDOCAM)

  4. Call to the Celestial Field Orientation Attunement Utility (CFOAU)

  5. Call to the Emotional State, Stress & Wave Form Clearing/Reprocess (ESSWFC/R) Utility

  6. Clearing & Process Flow Instructions of Roots & Permissive Mind/Body States

  7. Initial Client/Host Epigenome Session Scan

  8. Additional Session Scans

  9. Initial Epigenetic Discrepancies Session Scan

  10. Nucleosome Properties Addressed

  11. Critical Chromatin Structures Modulated

  12. Optimization of Chromatin Structures

  13. DNA Methylation & Related Modulation

  14. Nutritional Support Factors

  15. Histone Modification Modulation

  16. Client/Host DNA/Histone Relationship Identification Scan

  17. DNA/Histone Interaction History Registry Scan

  18. Identification of Applicable Histone Modification Proteins and Related Methyl Groups

  19. Histone Specific Base A & C Methylation Modification

  20. Linker Histone H1 Nucleosome Regulation Supported

  21. Histone H1 Role in Genomic Stabilization and Cancer Protection

  22. Particular Histone (HDAC5) Positive Selective Effects

  23. Noncoding DNA Factors Addressed

  24. Epigenetic Mechanism’s Dual Nature

  25. Accommodation of Two Transmission Routes

  26. Epigenetic Signal Regulation of New Brain Cells

  27. Transposable DNA Element (TE’s) Functions

  28. Identification of Mutant Alleles & Base Pair Substitutions

  29. Chromosomal Mutations Structure, Function and Inheritance

  30. Feedback Loops Link Energy & its Information to RNA-Mediated Protein Folding

  31. Virus-Driven Energy Theft & mRNA Degradation

  32. RNA Interference (RNAi) & Chromosomal Rearrangements

  33. RNA Interference (RNAi) & RNA Modulated System Stability

  34. Nutrient Energy Dependent Endogenous DNA Expression

  35. RNAi Gene Silencing & Repair

  36. Energy-Dependent Cell Type Differentiation

  37. Micro RNA (miRNA) Inhibition of DNA Expression

  38. MicroRNA (miRNA), Chromatin & DNA Organization

  39. Role of Small Interfering RNA (siRNA)

  40. Untranslated Regions & miRNA’s/piRNA’s

  41. MiRNA Processing/RNA & DNA Silencing

  42. Down-Regulation of Genes Specifically

  43. Up-Regulation of Genes Specifically

  44. NMD Control & Modulation of DNA Expression

  45. Placing & Monitoring Methyl Groups on DNA

  46. Necessary Removal of Methyl Tags from DNA

  47. RNA-Modification Therapeutic Application

  48. Olfactory Effects on Gene Expression & Immune System Function

  49. Entire Nervous System Interface

  50. Role of Hsp90 Protein & Prions

  51. FKPB5 Gene Trauma Risk Allele & Methylation

  52. Common MTHFR Gene Mutation Compensation

  53. Role of UHRF1 in Genetic Functions, Methylation & DNA Repair

  54. Kinesin-3 motor Unc-104 Signaling Mediation of Mutant Synaptic Defects Modulated

  55. Other Pernicious Gene Mutations & Genetic Disorders

  56. Microbiome Effects upon DNA Transcription & Chronic Disease

  57. Microbiome Effects on Client/Host Agency

  58. Viral/Bacterial Onset & Residual Effects

  59. Polycyclic Synthetic Effects Addressed & Modulated

  60. Functional Mechanisms of the Glucocorticoid Receptor (GR)

  61. GR-Dependent Gene Transcription

  62. Genetic Algorithm Gene Encoding & mRNA Splice Variants

  63. Types of Glucocorticoid Responsiveness (GR)

  64. MicroRNA Involvement in Regulation of GR Expression

  65. Periphery & CNS Involvement with GR & Depression

  66. GR Environment Signaling & Function Homeostasis Maintenance

  67. Specific Astrocyte/Microglial/TREM2 Genetic Issues

  68. The Circadian Rhythm Clockwork Mechanism Optimized & Modulated

  69. Maternal Care & Other Early Life Epigenetic Effects

  70. Identification & Rectification of Infantile Trauma Induced DNA/Epigenetic Alterations

  71. Sensory, Mental & Emotional Perception Affects & Effects

  72. Energetic Signatures & Stealth Information Session Scan

  73. Further DNA Damage Repair

  74. Genetic/Epigenetic Optimum Life Purpose Performance Scan

  75. Session Scan: Adam/Eve Covenant Considerations

  76. Deep Transgenerational Epigenetic Effects

  77. Call to Additional Modified/Extended Neuromodulation Pathways.

  78. Client/Host Epigenetic History Rectification

  79. Final Information Fault (IF) Translation for Clearing Purposes

  80. Final Data Processing Instructions

  81. Call to NG Behavioral & Habituation Change Path Process if Indicated

  82. Final Process Objectives Prep

  83. Final Clearing

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